Abstrakt

Przypisy

• 1. Afar B., Engel D., Clark E.A.: Activated lymphocyte subsets in adultperiodontitis. J. Periodontal. Res., 1992; 27: 126-133
  Google Scholar
• 2. Amer A., Singh G., Darke C., Dolby A.E.: Spontaneous lymphocyteproliferation in severe periodontal disease: role of T and B cells. J.Oral. Pathol. Med., 1990; 19: 49-52
  Google Scholar
• 3. Armitage G.C.: Development of a classification system for periodontaldiseases and conditions. Ann. Periodontol., 1999; 4: 1-6
  Google Scholar
• 4. Biron C.A., Nguyen K.B., Pien G.C., Cousens L.P., Salazar-MatherT.P.: Natural killer cells in antiviral defense: function and regulationby innate cytokines. Annu. Rev. Immunol., 1999; 17: 189-220
  Google Scholar
• 5. Boyton R.J., Altmann D.M.: Natural killer cells, killer immunoglobulin-likereceptors and human leucocyte antigen class I in disease.Clin. Exp. Immunol., 2007; 149: 1-8
  Google Scholar
• 6. Caligiuri M.A.: Human natural killer cells. Blood, 2008; 112: 461-469
  Google Scholar
• 7. Campbell K.S., Purdy A.K.: Structure/function of human killer cellimmunoglobulin-like receptors: lessons from polymorphisms, evolution,crystal structures and mutations. Immunology, 2011; 132: 315-325
  Google Scholar
• 8. Carrington M., Martin M.P.: The impact of variation at the KIRgene cluster on human disease. Curr. Top. Microbiol. Immunol.,2006; 298: 225-257
  Google Scholar
• 9. Chaushu S., Wilensky A., Gur C., Shapira L., Elboim M., Halftek G.,Polak D., Achdout H., Bachrach G., Mandelboim O.: Direct recognitionof Fusobacterium nucleatum by the NK cell natural cytotoxicityreceptor NKp46 aggravates periodontal disease. PLoS Pathog.,2012; 8: e1002601
  Google Scholar
• 10. Cobb C.M., Singla O., Feil P.H., Theisen F.C., Schultz R.E.: Comparisonof NK-cell (Leu-7+ and Leu-11b+) populations in clinicallyhealthy gingiva, chronic gingivitis and chronic adult periodontitis.J. Periodontal. Res., 1989; 24: 1-7
  Google Scholar
• 11. Cobb C.M., Singla O., Theisen F.C., Shultz R.E.: Ultrastructuralevidence of large granular lymphocyte (LGL) activity in lesions ofchronic adult periodontitis. J. Clin. Periodontol., 1990; 17: 371-378
  Google Scholar
• 12. Conigliaro P., Scrivo R., Valesini G., Perricone R.: Emerging rolefor NK cells in the pathogenesis of inflammatory arthropathies.Autoimmun. Rev., 2011; 10: 577-581
  Google Scholar
• 13. Fujita S., Takahashi H., Okabe H., Ozaki Y., Hara Y., Kato I.: Distributionof natural killer cells in periodontal diseases: an immunohistochemicalstudy. J. Periodontol., 1992; 63: 686-689
  Google Scholar
• 14. Garlet G.P., Martins W.Jr., Ferreira B.R., Milanezi C.M., Silva J.S.:Patterns of chemokines and chemokine receptors expression indifferent forms of human periodontal disease. J. Periodontal. Res.,2003; 38: 210-217
  Google Scholar
• 15. Ham H., Billadeau D.D.: Human immunodeficiency syndromesaffecting human natural killer cell cytolytic activity. Front. Immunol.,2014; 5: 2
  Google Scholar
• 16. Khakoo S.I., Carrington M.: KIR and disease: a model system orsystem of models? Immunol. Rev., 2006; 214, 186-201
  Google Scholar
• 17. Komiyama K., Hirsch H.Z., Moro I., Umemura S., Mestecky J.:HNK-1+ (Leu-7) cells and natural killer cell activity in inflamed humangingival tissue. J. Oral. Pathol., 1988; 17: 118-123
  Google Scholar
• 18. Krämer B., Kebschull M., Nowak M., Demmer R.T., Haupt M.,Körner C., Perner S., Jepsen S., Nattermann J., Papapanou P.N.: Roleof the NK cell-activating receptor CRACC in periodontitis. InfectImmun., 2013; 81: 690-696
  Google Scholar
• 19. Kuśnierczyk P.: Killer cell immunoglobulin-like receptor geneassociations with autoimmune and allergic diseases, recurrent spontaneousabortion, and neoplasms. Front. Immunol., 2013; 4: 8
  Google Scholar
• 20. Laine M.L., Crielaard W., Loos B.G.: Genetic susceptibility toperiodontitis. Periodontol. 2000, 2012; 58: 37-68
  Google Scholar
• 21. Lange D.E., Plagmann H.C., Eenboom A., Promesberger A.:Klinische Bewertungsverfahren zur Objectivierung der Mundhygiene.Dtsch. Zahnärztl. Z., 1977; 32: 44-49
  Google Scholar
• 22. Lanier L.L.: NK cell recognition. Annu. Rev. Immunol., 2005;23: 225-274
  Google Scholar
• 23. Majorczyk E., Pawlik A., Łuszczek W., Nowak I., Wiśniewski A.,Jasek M., Kuśnierczyk P.: Associations of killer cell immunoglobulin-likereceptor genes with complications of rheumatoid arthritis.Genes Immun., 2007; 8: 678-683
  Google Scholar
• 24. Martelli F.S., Mengoni A., Martelli M., Rosati C., Fanti E.: VDRTaqI polymorphism is associated with chronic periodontitis in Italianpopulation. Arch. Oral. Biol., 2011; 56: 1494-1498
  Google Scholar
• 25. Muthukuru M.: Technical advance: decreased helper T cells andincreased natural killer cells in chronic periodontitis analyzed bya novel method for isolating resident lymphocytes. J. Leukoc. Biol.,2012; 92: 683-692
  Google Scholar
• 26. Mühlemann H.R., Son S.: Gingival sulcus bleeding – a leadingsymptom in initial gingivitis. Helv. Odontol. Acta, 1971; 15: 107-113
  Google Scholar
• 27. Niepiekło-Miniewska W., Majorczyk E., Matusiak Ł., GendzekhadzeK., Nowak I., Narbutt J., Lesiak A., Kuna P., Ponińska J., Pietkiewicz-SworowskaA., Samoliński B., Płoski R., Szepietowski J.C.,Senitzer D., Kuśnierczyk P.: Protective effect of the KIR2DS1 gene inatopic dermatitis. Gene, 2013; 527: 594-600
  Google Scholar
• 28. Parham P.: MHC class I molecules and KIRs in human history,health and survival. Nat. Rev. Immunol., 2005; 5: 201-214
  Google Scholar
• 29. Parham P., Norman P.J., Abi-Rached L., Guethlein L.A.: Human–specific evolution of killer cell immunoglobulin-like receptor recognitionof major histocompatibility complex class I molecules.Philos. Trans. R. Soc. Lond. B Biol. Sci., 2012; 367: 800-811
  Google Scholar
• 30. Pihlstrom B.L., Michalowicz B.S., Johnson N.W.: Periodontal diseases.Lancet, 2005; 366: 1809-1820
  Google Scholar
• 31. Reichert T., De Bruyère M., Deneys V., Tötterman T., LydyardP., Yuksel F., Chapel H., Jewell D., Van Hove L., Linden J., et al. Lymphocytesubset reference ranges in adult Caucasians. Clin. Immunol.Immunopathol., 1991; 60: 190-208
  Google Scholar
• 32. Seymour G.J., Gemmell E., Reinhardt R.A., Eastcott J., TaubmanM.A.: Immunopathogenesis of chronic inflammatory periodontaldisease: cellular and molecular mechanisms. J. Periodontal. Res.,1993; 28: 478-486
  Google Scholar
• 33. Sharma R., Das A.: Organ-specific phenotypic and functionalfeatures of NK cells in humans. Immunol. Res., 2014; 58: 125-131
  Google Scholar
• 34. Stelin S., Ramakrishan H., Talwar A., Arun K.V., Kumar T.S.:Immunohistological analysis of CD1a+ langerhans cells and CD57+natural killer cells in healthy and diseased human gingival tissue:a comparative study. J. Indian. Soc. Periodontol., 2009; 13: 150-154
  Google Scholar
• 35. Tsuchiya N., Kyogoku C., Miyashita R., Kuroki K.: Diversity ofhuman immune system multigene families and its implication inthe genetic background of rheumatic diseases. Curr. Med. Chem.,2007; 14: 431-439
  Google Scholar
• 36. Van Dyke T.E., Lester M.A., Shapira L.: The role of the host responsein periodontal disease progression: implications for futuretreatment strategies. J. Periodontol., 1993; 64: 792-806
  Google Scholar
• 37. Wiśniewski A., Jankowska R., Passowicz-Muszyńska E., WiśniewskaE., Majorczyk E., Nowak I., Frydecka I., Kuśnierczyk P.: KIR2DL2/S2 and HLA-C C1C1 genotype is associated with better response totreatment and prolonged survival of patients with non-small celllung cancer in a Polish Caucasian population. Hum. Immunol., 2012;73: 927-931
  Google Scholar
• 38. Yamazaki K., Ohsawa Y., Yoshie H.: Elevated proportion of naturalkiller T cells in periodontitis lesions: a common feature ofchronic inflammatory diseases. Am. J. Pathol., 2001; 158: 1391-1398
  Google Scholar

Pełna treść artykułu