Abstrakt

Przypisy

• 1. Allavena P., Sica A., Vecchi A., Locati M., Sozzani S., Mantovani A.:The chemokine receptor switch paradigm and dendritic cell migration:its significance in tumor tissues. Immunol. Rev., 2000; 177: 141-149
  Google Scholar
• 2. Alvarez M.M., Liu J.C., Santiago G.T., Cha B.H., Vishwakarma A.,Ghaemmaghami AM., Khademhosseini A.: Delivery strategies tocontrol inflammatory response: modulating M1-M2 polarization intissue engineering applications. J. Control. Release, 2016; pii: S0168-3659(16)30023-2
  Google Scholar
• 3. Anthony R.M., Rutitzky L.I., Urban J.F.Jr., Stadecker M.J., GauseW.C.: Protective immune mechanisms in helminth infection. Nat.Rev. Immunol., 2007; 7: 975-987
  Google Scholar
• 4. Arend W.P., Malyak M., Guthridge C.J., Gabay C.: Interleukin-1receptor antagonist: role in biology. Annu. Rev. Immunol., 1998;16: 27-55
  Google Scholar
• 5. Bédard P.A., Golds E.E.: Cytokine-induced expression of mRNAsfor chemotactic factors in human synovial cells and fibroblasts. J.Cell. Physiol., 1993; 154: 433-441
  Google Scholar
• 6. Blank M., Gershwin M.E.: Autoimmunity: from the mosaic to thekaleidoscope. J. Autoimmun., 2008; 30: 1-4
  Google Scholar
• 7. Bogdan C., Paik J., Vodovotz Y., Nathan C.: Contrasting mechanismsfor suppression of macrophage cytokine release by transforminggrowth factor-β and interleukin-10. J. Biol. Chem., 1992;267: 23301-23308
  Google Scholar
• 8. Bosco M.C., Delfino S., Ferlito F., Battaglia F., Puppo M., GregorioA., Gambini C., Gattorno M., Martini A., Varesio L.: Hypoxic synovialenvironment and expression of macrophage inflammatory protein3g/CCL20 in juvenile idiopathic arthritis. Arthritis Rheum., 2008;58: 1833-1838
  Google Scholar
• 9. Bucala R.: MIF rediscovered: cytokine, pituitary hormone, andglucocorticoid-induced regulator of the immune response. FASEBJ., 1996; 10: 1607-1613
  Google Scholar
• 10. Burmester G.R., Weinblatt M.E., McInnes I.B., Porter D., BarbarashO., Vatutin M., Szombati I., Esfandiari E., Sleeman M.A., KaneC.D., Cavet G., Wang B., Godwood A., Magrini F., EARTH Study Group:Efficacy and safety of mavrilimumab in subjects with rheumatoidarthritis. Ann. Rheum. Dis., 2013; 72: 1445-1452
  Google Scholar
• 11. Calandra T., Bernhagen J., Metz C.N., Spiegel L.A., Bacher M.,Donnelly T., Cerami A., Bucala R.: MIF as a glucocorticoid-inducedmodulator of cytokine production. Nature, 1995; 377: 68-71
  Google Scholar
• 12. Calandra T., Bernhagen J., Mitchell R.A., Bucala R.: The macrophageis an important and previously unrecognized source of macrophagemigration inhibitory factor. J. Exp. Med., 1994; 179: 1895-1902
  Google Scholar
• 13. Chieppa M., Bianchi G., Doni A., Del Prete A., Sironi M., LaskarinG., Monti P., Piemonti L., Biondi A., Mantovani A., Introna M., AllavenaP.: Cross-linking of the mannose receptor on monocyte-deriveddendritic cells activates an anti-inflammatory immunosuppressiveprogram. J. Immunol., 2003; 171: 4552-4560
  Google Scholar
• 14. Chomarat P., Rissoan M.C., Pin J.J., Banchereau J., Miossec P.:Contribution of IL-1, CD14, and CD13 in the increased IL-6 productioninduced by in vitro monocyte-synoviocyte interactions. J. Immunol.,1995; 155: 3645-3652
  Google Scholar
• 15. Codarri L., Gyülvészi G., Tosevski V., Hesske L., Fontana A., MagnenatL., Suter T., Becher B.: RORγt drives production of the cytokineGM-CSF in helper T cells, which is essential for the effector phase ofautoimmune neuroinflammation. Nat. Immunol., 2011; 12: 560-567
  Google Scholar
• 16. Cook A.D., Turner A.L., Braine E.L., Pobjoy J., Lenzo J.C., HamiltonJ.A.: Regulation of systemic and local myeloid cell subpopulationsby bone marrow cell-derived granulocyte–macrophage colonystimulatingfactor in experimental inflammatory arthritis. ArthritisRheum., 2011; 63: 2340-2351
  Google Scholar
• 17. Cornish A.L., Campbell I.K., McKenzie B.S., Chatfield S., WicksI.P.: G-CSF and GM-CSF as therapeutic targets in rheumatoid arthritis.Nat. Rev. Rheumatol., 2009; 5: 554-559
  Google Scholar
• 18. Dayer J.M., Fenner H.: The role of cytokines and their inhibitorsin arthritis. Baillieres Clin. Rheumatol., 1992; 6: 485-516
  Google Scholar
• 19. De Benedetti F., Pignatti P., Massa M., Sartirana P., Ravelli A., CassaniG., Corti A., Martini A.: Soluble tumour necrosis factor receptorlevels reflect coagulation abnormalities in systemic juvenile chronicarthritis. Br. J. Rheumatol., 1997; 36: 581-588
  Google Scholar
• 20. De Benedetti F., Ravelli A., Martini A.: Cytokines in juvenile rheumatoidarthritis. Curr. Opin. Rheumatol., 1997; 9: 428-433
  Google Scholar
• 21. de Jager W., Vastert S.J., Beekman J.M., Wulffraat N.M., Kuis W.,Coffer P.J., Prakken B.J.: Defective phosphorylation of interleukin-18receptor β causes impaired natural killer cell function in systemic-onsetjuvenile idiopathic arthritis. Arthritis Rheum., 2009; 60: 2782-2793
  Google Scholar
• 22. Distler J.H., Wenger R.H., Gassmann M., Kurowska M., Hirth A.,Gay S., Distler O.: Physiologic responses to hypoxia and implicationsfor hypoxia-inducible factors in the pathogenesis of rheumatoidarthritis. Arthritis Rheum., 2004; 50: 10-23
  Google Scholar
• 23. El Kasmi K.C., Qualls J.E., Pesce J.T., Smith A.M., Thompson R.W.,Henao-Tamayo M., Basaraba R.J., König T., Schleicher U., Koo M.S.,Kaplan G., Fitzgerald K.A., Tuomanen E.I., Orme I.M., KannegantiT.D., Bogdan C., Wynn T.A., Murray P.J.: Toll-like receptor-inducedarginase 1 in macrophages thwarts effective immunity against intracellularpathogens. Nat. Immunol., 2008; 9: 1399-1406
  Google Scholar
• 24. El-Behi M., Ciric B., Dai H., Yan Y., Cullimore M., Safavi F., ZhangG.X., Dittel B.N., Rostami A.: The encephalitogenicity of TH17 cells isdependent on IL-1- and IL-23-induced production of the cytokineGM-CSF. Nat. Immunol., 2011; 12: 568-575
  Google Scholar
• 25. Fairweather D., Frisancho-Kiss S., Rose N.R.: Sex differences inautoimmune disease from a pathological perspective. Am. J. Pathol.,2008; 173: 600-609
  Google Scholar
• 26. Fairweather D., Frisancho-Kiss S., Yusung S.A., Barrett M.A.,Davis S.E., Gatewood S.J., Njoku D.B., Rose N.R.: Interferon-g protectsagainst chronic viral myocarditis by reducing mast cell degranulation,fibrosis, and the profibrotic cytokines transforminggrowth factor-b1, interleukin-1b, and interleukin-4 in the heart.Am. J. Pathol., 2004; 165: 1883-1894
  Google Scholar
• 27. Feldmann M., Brennan F.M., Maini R.N.: Role of cytokines inrheumatoid arthritis. Annu. Rev. Immunol., 1996; 14: 397-440
  Google Scholar
• 28. Firestein G.S.: Evolving concepts of rheumatoid arthritis. Nature,2003; 423: 356-361
  Google Scholar
• 29. Frisancho-Kiss S., Davis S.E., Nyland J.F., Frisancho J.A., CihakovaD., Barrett M.A., Rose N.R., Fairweather D.: Cutting edge. Cross-regulationby TLR4 and T cell Ig mucin-3 determines sex differencesin inflammatory heart disease. J. Immunol., 2007; 178: 6710-6714
  Google Scholar
• 30. Gattorno M., Facchetti P., Ghiotto F., Vignola S., BuoncompagniA., Prigione I., Picco P., Pistoia V.: Synovial fluid T cell clones fromoligoarticular juvenile arthritis patients display a prevalent Th1/Th0-type pattern of cytokine secretion irrespective of immunophenotype.Clin. Exp. Immunol., 1997; 109: 4-11
  Google Scholar
• 31. Giatromanolaki A., Siviridis E., Maltezos E., Athanassou N., PapazoglouD., Gatter K.C., Harris A.L., Koukourakis M.I.: Upregulatedhypoxia inducible factor-1α and -2α pathway in rheumatoid arthritisand osteoarthritis. Arthritis Res. Ther., 2003; 5: R193-R201
  Google Scholar
• 32. Gracie J.A.: Interleukin-18 as a potential target in inflammatoryarthritis. Clin. Exp. Immunol., 2004; 136: 402-440
  Google Scholar
• 33. Gracie J.A., Robertson S.E., McInnes I.B.: Interleukin-18. J. Leukoc.Biol., 2003; 73: 213-224
  Google Scholar
• 34. Grom A.A.: Natural killer cell dysfunction: a common pathwayin systemic-onset juvenile rheumatoid arthritis, macrophage activationsyndrome, and hemophagocytic lymphohistiocytosis? ArthritisRheum., 2004; 50: 689-698
  Google Scholar
• 35. Grom A.A., Villanueva J., Lee S., Goldmuntz E.A., Passo M.H.,Filipovich A.: Natural killer cell dysfunction in patients with systemic-onsetjuvenile rheumatoid arthritis and macrophage activationsyndrome. J. Pediatr., 2003; 142: 292-296
  Google Scholar
• 36. Haringman J.J., Smeets T.J., Reinders-Blankert P., Tak P.P.: Chemokineand chemokine receptor expression in paired peripheralblood mononuclear cells and synovial tissue of patients with rheumatoidarthritis, osteoarthritis, and reactive arthritis. Ann. Rheum.Dis., 2006; 65: 294-300
  Google Scholar
• 37. Henderson B., Pettipher E.R.: Arthritogenic actions of recombinantIL-1 and tumour necrosis factor α in the rabbit: evidence forsynergistic interactions between cytokines in vivo. Clin. Exp. Immunol.,1989; 75: 306-310
  Google Scholar
• 38. Houssiau F.A., Devogelaer J.P., Van Damme J., de DeuxchaisnesC.N., Van Snick J.: Interleukin-6 in synovial fluid and serum of patientswith rheumatoid arthritis and other inflammatory arthritides.Arthritis Rheum., 1988; 31: 784-788
  Google Scholar
• 39. Jelusić M., Lukić I.K., Tambić-Bukovac L., Dubravcić K., Malcić I.,Rudan I., Batinić D.: Interleukin-18 as a mediator of systemic juvenileidiopathic arthritis. Clin. Rheumatol., 2007; 26: 1332-1334
  Google Scholar
• 40. Juhas U., Ryba-Stanisławowska M., Szargiej P., Myśliwska J.: Differentpathways of macrophage activation and polarization. PostępyHig. Med. Dośw., 2015; 69: 496-502
  Google Scholar
• 41. Kasama T., Ohtsuka K., Sato M., Takahashi R., Wakabayashi K.,Kobayashi K.: Macrophage migration inhibitory factor: a multifunctionalcytokine in rheumatic diseases. Arthritis, 2010; 2010: 106202
  Google Scholar
• 42. Kerrigan A.M., Brown G.D.: C-type lectins and phagocytosis. Immunobiology,2009; 214: 562-575
  Google Scholar
• 43. Koch A.E.: Chemokines and their receptors in rheumatoid arthritis:future targets? Arthritis Rheum., 2005; 52: 710-721
  Google Scholar
• 44. Koch A.E., Polverini P.J., Kunkel S.L., Harlow L.A., DiPietro L.A,Elner V.M., Elner S.G., Strieter R.M.: Interleukin-8 as a macrophagederivedmediator of angiogenesis. Science, 1992; 258: 1798-1801
  Google Scholar
• 45. Kotake S., Sato K., Kim K.J., Takahashi N., Udagawa N., NakamuraI., Yamaguchi A., Kishimoto T., Suda T., Kashiwazaki S.: Interleukin-6and soluble interleukin-6 receptors in the synovial fluids from rheumatoidarthritis patients are responsible for osteoclast-like cell formation.J. Bone Miner. Res., 1996; 11: 88-95
  Google Scholar
• 46. Lassmann H., Brück W., Lucchinetti C.: Heterogeneity of multiplesclerosis pathogenesis: implications for diagnosis and therapy.Trends Mol. Med., 2001; 7: 115-121
  Google Scholar
• 47. Lee D.M., Weinblatt M.E.: Rheumatoid arthritis. Lancet, 2001;358: 903-911
  Google Scholar
• 48. Leech M., Metz C., Hall P., Hutchinson P., Gianis K., Smith M.,Weedon H., Holdsworth S.R., Bucala R., Morand E.F.: Macrophagemigration inhibitory factor in rheumatoid arthritis: evidence of proinflammatoryfunction and regulation by glucocorticoids. ArthritisRheum., 1999; 42: 1601-1608
  Google Scholar
• 49. Leech M., Metz C., Santos L., Peng T., Holdsworth S.R., BucalaR., Morand E.F.: Involvement of macrophage migration inhibitoryfactor in the evolution of rat adjuvant arthritis. Arthritis Rheum.,1998; 41: 910-917
  Google Scholar
• 50. Ling X.B., Macaubas C., Alexander H.C., Wen Q., Chen E., PengS., Sun Y., Deshpande C., Pan K.H., Lin R., Lih C.J., Chang S.Y., LeeT., Sandborg C., Begovich A.B., Cohen S.N., Mellins E.D.: Correlationanalyses of clinical and molecular findings identify candidatebiological pathways in systemic juvenile idiopathic arthritis. BMCMed., 2012; 10: 125
  Google Scholar
• 51. Loetscher P., Dewald B., Baggiolini M., Seitz M.: Monocyte chemoattractant protein 1 and interleukin 8 production by rheumatoidsynoviocytes. Effects of anti-rheumatic drugs. Cytokine, 1994;6: 162-170
  Google Scholar
• 52. Lotito A.P., Campa A., Silva C.A., Kiss M.H., Mello S.B.: Interleukin 18 as a marker of disease activity and severity in patients withjuvenile idiopathic arthritis. J. Rheumatol., 2007; 34: 823-830
  Google Scholar
• 53. Ma Y., Pope R.M.: The role of macrophages in rheumatoid arthritis.Curr. Pharm. Des., 2005; 11: 569-580
  Google Scholar
• 54. Mackay I.R., Leskovsek N.V., Rose N.R.: Cell damage and autoimmunity:a critical appraisal. J. Autoimmun., 2008; 30: 5-11
  Google Scholar
• 55. Martinez F.O., Helming L., Gordon S.: Alternative activation ofmacrophages: an immunologic functional perspective. Annu. Rev.Immunol., 2009; 27: 451-483
  Google Scholar
• 56. Martinez F.O., Sica A., Mantovani A., Locati M.: Macrophage activationand polarization. Front. Biosci., 2008; 13: 453-461
  Google Scholar
• 57. Meazza C., Travaglino P., Pignatti P., Magni-Manzoni S., RavelliA., Martini A., De Benedetti F.: Macrophage migration inhibitory factorin patients with juvenile idiopathic arthritis. Arthritis Rheum.,2002; 46: 232-237
  Google Scholar
• 58. Mikulowska A., Metz C.N., Bucala R., Holmdahl R.: Macrophagemigration inhibitory factor is involved in the pathogenesis of collagentype II-induced arthritis in mice. J. Immunol., 1997; 158: 5514-5517
  Google Scholar
• 59. Murray P.J., Wynn T.A.: Protective and pathogenic functions ofmacrophage subsets. Nat. Rev. Immunol., 2011; 11: 723-737
  Google Scholar
• 60. Müssener A., Klareskog L., Lorentzen J.C., Kleinau S.: TNF-αdominates cytokine mRNA expression in lymphoid tissues of ratsdeveloping collagen- and oil-induced arthritis. Scand. J. Immunol.,1995; 42: 128-134
  Google Scholar
• 61. Nazimek K., Filipczak-Bryniarska I., Bryniarski K.: The role ofmedicaments, exosomes and miRNA molecules in modulation ofmacrophage immune activity. Postępy Hig. Med. Dośw., 2015; 69:1114-1129
  Google Scholar
• 62. Neidel J., Schulze M., Lindschau J.: Association between degreeof bone-erosion and synovial fluid-levels of tumor necrosis factorα in the knee-joints of patients with rheumatoid arthritis. Inflamm.Res., 1995; 44: 217-221
  Google Scholar
• 63. Nickel T., Schmauss D., Hanssen H., Sicic Z., Krebs B., Jankl S.,Summo C., Fraunberger P., Walli A.K., Pfeiler S., Weis M.: oxLDL uptakeby dendritic cells induces upregulation of scavenger-receptors,maturation and differentiation. Atherosclerosis, 2009; 205: 442-450
  Google Scholar
• 64. Nirmala N., Grom A., Gram H.: Biomarkers in systemic juvenileidiopathic arthritis: a comparison with biomarkers in cryopyrinassociatedperiodic syndromes. Curr. Opin. Rheumatol., 2014; 26:543-552
  Google Scholar
• 65. Okamura H., Nagata K., Komatsu T., Tanimoto T., Nukata Y.,Tanabe F., Akita K., Torigoe K., Okura T., Fukuda S., Kurimoto M.:A novel costimulatory factor for gamma interferon induction foundin the livers of mice causes endotoxic shock. Infect. Immun., 1995;63: 3966-3972
  Google Scholar
• 66. Pelletier M., Maggi L., Micheletti A., Lazzeri E., Tamassia N.,Costantini C., Cosmi L., Lunardi C., Annunziato F., Romagnani S., CassatellaM.A.: Evidence for a cross-talk between human neutrophilsand Th17 cells. Blood, 2010; 115: 335-343
  Google Scholar
• 67. Petty R.E., Southwood T.R., Manners P., Baum J., Glass D.N., GoldenbergJ., He X., Maldonado-Cocco J., Orozco-Alcala J., Prieur A.M.,Suarez-Almazor M.E., Woo P., International League of Associationsfor Rheumatology: International League of Associations for Rheumatologyclassification of juvenile idiopathic arthritis: second revision,Edmonton, 2001. J. Rheumatol., 2004; 31: 390-392
  Google Scholar
• 68. Piper C., Pesenacker A.M., Bending D., Thirugnanabalan B.,Varsani H., Wedderburn L.R., Nistala K.: T cell expression of granulocyte-macrophagecolony-stimulating factor in juvenile arthritis is contingent upon Th17 plasticity. Arthritis Rheumatol., 2014; 66:1955-1960
  Google Scholar
• 69. Ponomarev E.D., Maresz K., Tan Y., Dittel B.N.: CNS-derived interleukin-4is essential for the regulation of autoimmune inflammationand induces a state of alternative activation in microglial cells.J. Neurosci., 2007; 27: 10714-10721
  Google Scholar
• 70. Prakken B., Albani S., Martini A.: Juvenile idiopathic arthritis.Lancet, 2011; 377: 2138-2149
  Google Scholar
• 71. Probert L., Plows D., Kontogeorgos G., Kollias G.: The type I interleukin-1receptor acts in series with tumor necrosis factor (TNF) to inducearthritis in TNF-transgenic mice. Eur. J. Immunol., 1995; 25: 1794-1797
  Google Scholar
• 72. Ravelli A., Martini A.: Juvenile idiopathic arthritis. Lancet, 2007;369: 767-778
  Google Scholar
• 73. Schädlich H., Ermann J., Biskop M., Falk W., Sperling F., JüngelA., Lehmann J., Emmrich F., Sack U.: Anti-inflammatory effects ofsystemic anti-tumour necrosis factor α treatment in human/murineSCID arthritis. Ann. Rheum. Dis., 1999; 58: 428-434
  Google Scholar
• 74. Schmidt-Weber C.B., Pohlers D., Siegling A., Schädlich H., BuchnerE., Volk H.D., Palombo-Kinne E., Emmrich F., Kinne R.W.: Cytokinegene activation in synovial membrane, regional lymph nodes, andspleen during the course of rat adjuvant arthritis. Cell. Immunol.,1999; 195: 53-65
  Google Scholar
• 75. Schutyser E., Struyf S., Van Damme J.: The CC chemokine CCL20and its receptor CCR6. Cytokine Growth Factor Rev., 2003; 14: 409-426
  Google Scholar
• 76. Semenza G.L.: Hypoxia-inducible factor 1: oxygen homeostasisand disease pathophysiology. Trends Mol. Med., 2001; 7: 345-350
  Google Scholar
• 77. Shoenfeld Y., Selmi C., Zimlichman E., Gershwin M.E.: The autoimmunologist:geoepidemiology, a new center of gravity, and primetime for autoimmunity. J. Autoimmmun., 2008; 31: 325-330
  Google Scholar
• 78. Sica A., Bronte V.: Altered macrophage differentiation and immunedysfunction in tumor development. J. Clin. Invest., 2007; 117:1155-1166
  Google Scholar
• 79. Stein M., Keshav S., Harris N., Gordon S.: Interleukin 4 potentlyenhances murine macrophage mannose receptor activity: a markerof alternative immunologic macrophage activation. J. Exp. Med.,1992; 176: 287-292
  Google Scholar
• 80. Stéphan J.L., Koné-Paut I., Galambrun C., Mouy R., Bader-MeunierB., Prieur A.M.: Reactive haemophagocytic syndrome in childrenwith inflammatory disorders. A retrospective study of 24 patients.Rheumatology, 2001; 40: 1285-1292
  Google Scholar
• 81. Van Ginderachter J.A., Movahedi K., Hassanzadeh GhassabehG., Meerschaut S., Beschin A., Raes G., De Baetselier P.: Classicaland alternative activation of mononuclear phagocytes: Picking thebest of both worlds for tumor promotion. Immunobiology, 2006;211: 487-501
  Google Scholar
• 82. Villanueva J., Lee S., Giannini E.H., Graham T.B., Passo M.H.,Filipovich A., Grom A.A.: Natural killer cell dysfunction is a distinguishingfeature of systemic onset juvenile rheumatoid arthritis andmacrophage activation syndrome. Arthritis Res. Ther., 2005; 7: 30-37
  Google Scholar
• 83. Wagner-Weiner L.: Systemic juvenile idiopathic arthritis complicatedby macrophage activation syndrome. Pediatr Ann., 2015;44: e142-e147
  Google Scholar
• 84. Wood N.C., Dickens E., Symons J.A., Duff G.W.: In situ hybridizationof interleukin-1 in CD14-positive cells in rheumatoid arthritis.Clin. Immunol. Immunopathol., 1992; 62: 295-300
  Google Scholar
• 85. Wood N.C., Symons J.A., Dickens E., Duff G.W.: In situ hybridizationof IL-6 in rheumatoid arthritis. Clin. Exp. Immunol., 1992;87: 183-189
  Google Scholar

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