Abstrakt

Przypisy

• 1. Cayatte A.J., Palacino J.J., Horten K., Cohen R.A.: Chronic inhibitionof nitric oxide production accelerates neointima formationand impairs endothelial function in hypercholesterolemic rabbits.Arterioscler. Thromb., 1994: 14: 753-759
  Google Scholar
• 2. Chilian W.M., Mass H.J., Williams S.E., Layne S.M., Smith E.E.,Scheel K.W.: Microvascular occlusions promote coronary collateralgrowth. Am. J. Physiol. 1990; 258: H1103-H1111
  Google Scholar
• 3. Chilian W.M., Penn M.S., Pung Y.F., Dong F., Mayorga M., OhanyanV., Logan S., Yin L.: Coronary collateral growth – back to the future.J. Mol. Cell. Cardiol., 2012; 52: 905-911
  Google Scholar
• 4. Colombo M.G., Paradossi U., Andreassi M.G., Botto N., ManfrediS., Masetti S., Biagini A., Clerico A.: Endothelial nitric oxide synthasegene polymorphisms and risk of coronary artery disease. Clin.Chem., 2003; 49: 389-395
  Google Scholar
• 5. Dosenko V.E., Zagoriy V.Y., Lutay Y.M., Parkhomenko A.N., MoibenkoA.A.: Allelic polymorphism in the promoter (T–>C), but notin exon 7 (G–>T) or the variable number tandem repeat in intron4, of the endothelial nitric oxide synthase gene is positively associatedwith acute coronary syndrome in the Ukrainian population.Exp. Clin. Cardiol., 2006; 11: 11-13
  Google Scholar
• 6. Fujita M., Ikemoto M., Kishishita M., Otani H., Nohara R., TanakaT., Tamaki S., Yamazato A., Sasayama S.: Elevated basic fibroblastgrowth factor in pericardial fluid of patients with unstable angina.Circulation, 1996; 94: 610-613
  Google Scholar
• 7. Gulec S., Karabulut H., Ozdemir A.O., Ozdol C., Turhan S., AltınT., Tutar E., Genc Y., Erol C.: Glu298Asp polymorphism of the eNOSgene is associated with coronary collateral development. Atherosclerosis,2008; 198: 354-359
  Google Scholar
• 8. Hood J.D., Meininger C.J., Ziche M., Granger H.J.: VEGF upregulatesecNOS message, protein, and NO production in human endothelialcells. Am. J. Physiol., 1998; 274: H1054-H1058
  Google Scholar
• 9. Hyndman M.E., Parsons H.G., Verma S., Bridge P.J., EdworthyS., Jones C., Lonn E., Charbonneau F., Anderson T.J.: The T-786–>Cmutation in endothelial nitric oxide synthase is associated withhypertension. Hypertension, 2002; 39: 919-922
  Google Scholar
• 10. Jeerooburkhan N., Jones L.C., Bujac S., Cooper J.A., Miller G.J.,Vallance P., Humphries S.E., Hingorani A.D.: Genetic and environmentaldeterminants of plasma nitrogen oxides and risk of ischemicheart disease. Hypertension, 2001; 38: 1054-1061
  Google Scholar
• 11. Lamblin N., Cuilleret F.J., Helbecque N., Dallongeville J., LablancheJ.M., Amouyel P., Bauters C., Van Belle E.: A common variant ofendothelial nitric oxide synthase (Glu298Asp) is associated with collateraldevelopment in patients with chronic coronary occlusions.BMC Cardiovasc. Disord., 2005; 5: 27
  Google Scholar
• 12. Levin D.C.: Pathways and functional significance of the coronarycollateral circulation. Circulation, 1974; 50: 831-837
  Google Scholar
• 13. Liu D., Jiang Z., Dai L., Zhang X., Yan C., Han Y.: Association between the – 786T>C 1 polymorphism in the promoter region of endothelialnitric oxide synthase (eNOS) and risk of coronary artery disease:a systematic review and meta-analysis. Gene, 2014; 545: 175-183
  Google Scholar
• 14. Ludmer P.L., Selwyn A.P., Shook T.L., Wayne R.R., Mudge G.H.,Alexander R.W., Ganz P.: Paradoxical vasoconstriction induced byacetylcholine in atherosclerotic coronary arteries. N. Engl. J. Med.,1986; 315: 1046-1051
  Google Scholar
• 15. Meier P., Heminway H., Lansky A.J., Knapp G., Pitt B., Seiler C.:The impact of the coronary collateral circulation on mortality: ameta-analysis. Eur. Heart J., 2012; 33: 614-621
  Google Scholar
• 16. Meier P., Schirmer S.H., Lansky A.J., Timmis A., Pitt B., SeilerC.: The collateral circulation of the heart. BMC Med., 2013; 11: 143
  Google Scholar
• 17. Metzger I.F., Souza-Costa D.C., Marroni A.S., Nagassaki S., DestaZ., Flockhart D.A., Tanus-Santos J.E.: Endothelial nitric oxide synthasegene haplotypes associated with circulating concentrationsof nitric oxide products in healthy men. Pharmacogenet. Genomics,2005; 15: 565-570
  Google Scholar
• 18. Moncada S., Higgs A.: The L-arginine-nitric oxide pathway. N.Engl. J. Med., 1993; 329: 2002-2012
  Google Scholar
• 19. Murohara T., Asahara T., Silver M., Bauters C., Masuda H., KalkaC., Kearney M., Chen D., Symes J.F., Fishman M.C., Huang P.L., IsnerJ.M.: Nitric oxide synthase modulates angiogenesis in response totissue ischemia. J. Clin. Invest., 1998; 101: 2567-2578
  Google Scholar
• 20. Nagassaki S., Metzger I.F., Souza-Costa D.C., Marroni A.S., UzuelliJ.A., Tanus-Santos J.E.: eNOS genotype is without effect on circulatingnitrite/nitrate level in healthy male population. Thromb.Res., 2005; 115: 375-379
  Google Scholar
• 21. Nakayama M., Yasue H., Yoshimura M., Shimasaki Y., KugiyamaK., Ogawa H., Motoyama T., Saito Y., Ogawa Y., Miyamoto Y., NakaoK.: T-786–>C mutation in the 5’-flanking region of the endothelialnitric oxide synthase gene is associated with coronary spasm. Circulation,1999; 99: 2864-2870
  Google Scholar
• 22. Nakayama M., Yasue H., Yoshimura M., Shimasaki Y., Ogawa H.,Kugiyama K., Mizuno Y., Harada E., Nakamura S., Ito T., Saito Y., MiyamotoY., Ogawa Y., Nakao K.: T-786–>C mutation in the 5’-flankingregion of the endothelial nitric oxide synthase gene is associatedwith myocardial infarction, especially without coronary organicstenosis. Am. J. Cardiol., 2000; 86: 628-634
  Google Scholar
• 23. Newman P.E.: The coronary collateral circulation: determinantsand functional significance in ischemic heart disease. Am. Heart J.,1981; 102: 431-445
  Google Scholar
• 24. Papapetropoulos A., García-Cardeña G., Madri J.A., Sessa WC.:Nitric oxide production contributes to the angiogenic propertiesof vascular endothelial growth factor in human endothelial cells. J.Clin. Invest.. 1997: 100: 3131-3139
  Google Scholar
• 25. Rentrop K.P., Cohen M., Blanke H., Phillips R.A.: Changes in collateralchannel filling immediately after controlled coronary artery occlusion by an angioplasty balloon in human subjects. J. Am. Coll.Cardiol., 1985; 5: 587-592
  Google Scholar
• 26. Rossi G.P., Cesari M., Zanchetta M., Colonna S., Maiolino G., PedonL., Cavallin M., Maiolino P., Pessina A.C.: The T-786C endothelialnitric oxide synthase genotype is a novel risk factor for coronaryartery disease in Caucasian patients of the GENICA Study. J. Am. Coll.Cardiol., 2003; 41: 930-937
  Google Scholar
• 27. Rossi G.P., Maiolino G., Zanchetta M., Sticchi D., Pedon L., CesariM., Montemurro D., De Toni R., Zavattiero S., Pessina A.C.: The T-786Cendothelial nitric oxide synthase genotype predicts cardiovascularmortality in high-risk patients. J. Am. Coll. Cardiol., 2006; 48: 1166-1174
  Google Scholar
• 28. Sabia P.J., Powers E.R., Ragosta M., Sarembock I.J., Burwell L.R.,Kaul S.: An association between collateral blood flow and myocardialviability in patients with recent myocardial infarction. N. Engl.J. Med., 1992; 327: 1825-1831
  Google Scholar
• 29. Schmidt H.H., Walter U.: NO at work. Cell, 1994; 78: 919-925
  Google Scholar
• 30. Tangürek B., Özer N., Sayar N., Terzi S., Yilmaz H., Asiltürk R.,Aksu H., Ciloglu F., Aksoy S., Cagil A.: The relationship between endothelialnitric oxide synthase gene polymorphism (T-786 C) andcoronary artery disease in a Turkish population. Arch. Turk. Soc.Cardiol., 2005; 33: 467-472
  Google Scholar
• 31. Tesauro M., Thompson W.C., Rogliani P., Qi L., Chaudhary P.P.,Moss J.: Intracellular processing of endothelial nitric oxide synthaseisoforms associated with differences in severity of cardiopulmonarydiseases: cleavage of proteins with aspartate vs. glutamate at position 298 Proc. Natl. Acad. Sci. USA, 2000; 97: 2832-2835
  Google Scholar
• 32. van der Zee R., Murohara T., Luo Z., Zollmann F., Passeri J., LekutatC., Isner J.M.: Vascular endothelial growth factor/vascular permeabilityfactor augments nitric oxide release from quiescent rabbitand human vascular endothelium. Circulation, 1997; 95: 1030-1037
  Google Scholar
• 33. Xiao M., Lu X., Li J., Li L., Li Y.: Physiologic ischaemic traininginduces endothelial progenitor cell mobilization and myocardialangiogenesis via endothelial nitric oxide synthase related pathwayin rabbits. J. Cardiovasc. Med., 2014; 15: 280-287
  Google Scholar
• 34. Ziche M., Morbidelli L., Choudhuri R., Zhang H.T., Donnini S.,Granger H.J., Bicknell R.: Nitric oxide synthase lies downstreamfrom vascular endothelial growth factor-induced but not basic fibroblastgrowth factor-induced angiogenesis. J. Clin. Invest., 1997;99: 2625-2634
  Google Scholar

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